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(B) Normal internal genital organs are visible, and there is no evidence of female genital organs on the T2-weighted magnetic resonance image. In this study, we present an SRY-negative 46,XX Korean boy who presented with small testes and without any genital ambiguity. Molecular genetic studies confirmed SOX9 duplication as the cause of 46,XX testicular DSD for the first time in Korea.

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Today’s Kinsey Institute is a dynamic interdisciplinary research center and archival collection, world-renowned for research, historical preservation, and education programs exploring sex, sexuality, relationships, and well-being. We welcome you to join us in the discovery of the unknown as we continue to advance knowledge and change the world for the better. For more than 75 years, the Kinsey Institute at Indiana University has been the trusted source for scientific knowledge and research on critical issues in sexuality, gender, and reproduction.

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46,XX male syndrome was first described in ), and the Chicago consensus renamed of this syndrome as 46,XX testicular DSD in ). Most patients have normal male phenotypes at birth, and are usually diagnosed in late adolescence because of delayed puberty, gynecomastia, or infertility2). Approximately 15% of XX males have hypospadias, cryptorchidism, or more severe genital ambiguity13). All 46,XX males are infertile owing to the absence of the azoospermia factor region in the long arm of the Y chromosome14,15).

• He reported in The Lancet that his vigor and feeling of well-being were markedly restored but the effects were transient,[221] and Brown-Séquard’s hopes for the compound were dashed.
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• Studies of laboratory pain provide additional evidence of hormonal influences on pain responses.
• Previous reviews have concluded that females are more sensitive to pain compared with males.127,327 The following review will determine whether more recent studies continue to support this conclusion.

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Cognitive/affective factors are important determinants of pain responses and likely contribute to sex differences in pain.289,335 The cognitive and affective mechanisms that have been investigated most widely in the context of sex and gender differences include coping processes, catastrophizing, and affective factors (eg, anxiety, depression). The following sections discuss the evidence regarding sex differences in the associations among psychological factors and pain report. A study of third- and fifth-grade children in Finland found that 32% reported a weekly musculoskeletal pain with significantly more girls reporting pain in chest (7%, 4%) and in the upper back (8%, 5%) compared with boys.278 Sex-related differences were not found for the lower back and neck pain.

Several studies have addressed whether physical or psychological interventions differentially influence pain responses in men versus women, and these studies are summarized in the lower portion of Table 10. The majority of these studies (5/6, 81%) indicate that females exhibit more efficient pain inhibitory responses compared with males. For example, using an isometric handgrip exercise, pressure pain threshold was elevated in females but not males, suggesting that exercise-induced analgesia was greater in females.226 However, sex-related differences in pain modulation may be dependent on the type of stressor and pain modality. For example, in response to a laboratory public-speaking stressor, males exhibited a greater stress-related reduction in heat pain, whereas females showed a greater reduction with ischemic pain.45 Sternberg et al387 reported that cold pain was reduced in males during video game competition, whereas pain was more substantially reduced by physical exercise in females.

The findings appear to indicate that females exhibited an increase in their pain tolerance when given the expectation that women would tolerate the pain for a longer time, whereas males showed similar pain tolerance regardless of expected tolerance time. Another study examined the effects of high versus low monetary incentives on pain tolerance among females and males, anticipating that high external incentives would produce stronger effects among females, because males possess higher endogenous motivation to tolerate pain.257 However, the incentive manipulation had no effect on pain tolerance for females or males. In addition to the above findings addressing sex differences in responses to μ-opioid agonists, others have investigated analgesic responses to mixed-action opioid agonist-antagonists among women relative to men.

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